The Dominoes fall the same with PSS from Lyme, GWS, ME, FMS, Vaccines.. Etc (Here’s how)..

Post-Sepsis Syndrome is literally the dominoes game from hell – like some twisting turning course that some kid spent hours setting up over the coffee table and down the basement stairs, poised for action, set and prepared by steady hands, but all it takes is one tiny movement of a finger, or for someone to slam the front door, to start the chain reaction, except in this case it’s not a finger, it’s an infection, a septic infection that will leave you staggering in a terrifying soup of sickness, a frightening fog you can’t escape no matter how many times you scream out for help, or how many tears you cry.  Help does NOT come, and not only does it not help when you meet it, it mocks you, rolls its eyes, laughs and sends you packing.

Now most people who deal with the commonly understood form of Sepsis are fighting for their lives in the ER or critical care unit of their local hospital.  And as serious as this is (and I’m not down playing it at all) in one way they are the lucky ones, for the simple fact that not only are they fighting for their lives, but the doctors are as well.  However there is another side to Sepsis that many of us experience, that doesn’t trigger as many warning bells (no being shuttled to the front of the triage list) and these extremely sick individuals are often left to suffer through agony and terror on their own AND at home.

This is a TLR 2/1 agonist type of Sepsis and its different from your common bacterial TLR 4 Sepsis.  The type of Sepsis I am talking about is caused by Triacylated Lipoproteins, and they are so toxic that they scramble the immune system, shutting it down and causing tolerance and cross tolerance as well as immune suppression that is permanent!   The alarm bells in this situation are certainly going off in the body but unfortunately often don’t cause the same urgency as family and friends usually assume it’s a bad bug etc and they’d be right, just not in the way they expected – these are STEALTH infections; and they do cause an acute reaction initially and certainly cause a lot of damage over time, but because of their unique interaction with the immune system they have found a way to stay, and stay for good while they collect a raft of fierce friends to surround them leaving you to  spend your life in a toxic soup of illness (But hey you look normal so nobody takes you seriously). Here’s my analogy; you can take a direct flight to Greece or you can go via Ontario and London but either way you end up in Greece, and either way you end up with Post Sepsis. Talk about injustice heaped upon injustice, but this would be nothing compared to the response to it.  It’s the response that get’s me; because as hard and hellish as it is to live with PSS, the response can be harder, because Immune Suppressed groups are the most abused groups in medicine.  “You are not DEpressed you are Oppressed” to quote Kathleen Dickson

So the good news is you lived through the Septic event, because after continuous exposure of TLR 2/1 agonists by OspA with Lyme and other Triacyl Lipoproteins from infections like Brucella, TB, other Spirochetal diseases and the slow growing Mycobacteria and Mycoplasmas, the immune system has wisely decided it best shut down or it’s going to be deads-ville for you.  Unfortunately in life if there’s an up side there’s generally also a down side and the immune system is no different; Meet the double-edged sword.

Novel Bacterial Lipoprotein Structures Conserved in Low-GC Content Gram-positive Bacteria Are Recognized by Toll-like Receptor 2

Bacterial lipoproteins/lipopeptides inducing host innate immune responses are sensed by mammalian Toll-like receptor 2 (TLR2). These bacterial lipoproteins are structurally divided into two groups, diacylated or triacylated lipoproteins, by the absence or presence of an amide-linked fatty acid.

Mycobacterium tuberculosis 19-kilodalton lipoprotein inhibits Mycobacterium smegmatis-induced cytokine production by human macrophages in vitro.

Lipoproteins are triacylated in slow-growing mycobacteria. BCG_2070c encodes a functional Lnt in M. bovis BCG. We identified mycobacteria-specific tuberculostearic acid as further substrate for N- acylation in slow-growing mycobacteria.

After being stealth bombed, the immune system becomes tolerized to the Triacyl Lipoproteins and has decided its best to turn a blind eye to them and ignore them all together. The up side to this is it prevents the immune system from over reacting and sending out an army of inflammatory cells that would probably kill you, the downside is you are now in a permanently immune suppressed state; the immune system is no longer mounting a response to the pathogen, and because the shut down was complete it will also ignore all sorts of other pathogens like viruses, bacteria and parasites and this is called cross tolerance.   The other big downside of this is you will likely not produce fever in the future (your temp will drop) so when your body is being ravaged by a cess-pool of infections you will once again not be setting off your every day alarm bells and once again you will be told your nuts and that it’s all in your head; when you break down and finally put a call in to the ambulance (which you would never do unless there were HARD odds on you dying) and are put at the bottom of the list at ER and cajoled into talking with a shrink while there……. Sigh…. the predictability of it and the utter waste of it too.

As a side note (to demonstrate the critical thinking skills of my GP) my then doctor told me that it was probably all in my head but that I was so seemingly sane and genuine that even a psychiatrist probably wouldn’t be able to detect it.  You see the problem here (If  you can’t tell there is a psychiatric problem and you don’t think a psychiatrist will be able to either why on earth would you think there was one?) Oh right cuz Mr God complex figures if he doesn’t know what it is it can’t possibly be real.  I remember thinking what the heck’s in your head doc lol, but I digress.

Sepsis and septic shock

“sepsis has become less of an immediate life-threatening disorder and more of a long-term chronic critical illness, often associated with prolonged inflammation, immune suppression, organ injury and lean tissue wasting. Furthermore, patients who survive sepsis have continuing risk of mortality after discharge, as well as long-term cognitive and functional deficits”

In order for something to gain acceptance in science other scientists must be able to reproduce the results in further experiments and the mechanism needs to be shown to exist in parallel.

Lyme disease (Borrelia Burgdorferi) is known to shed Osps (OspA and B etc) that are Triacyl Lipoproteins (TL).   These TL’s cause tolerance and cross tolerance which essentially detonates then paralyses the immune system causing reactivation of latent viruses and opportunistic infections.     If Triacyl Lipoproteins from Lyme cause TLR 2/1 tolerance and cross tolerance (to other TLR ‘s that manage other bacteria) they will also cause the same issues if shed by other types of bacteria.  This is how you show your mechanism in parallel.  So what other types of bacteria shed Triacyl Lipoproteins?  So far

Slow-growing Mycobactieria’s (Like leprosy; Mycobacterium lepromatosis and Tuberculosis; Mycobacterium Tuberculosis…..), Brucella, Mycoplasmas and other Spirochetal diseases such as Syphilis.  These are some of the most deadly infections, they cause a long, slow painful death and Lyme is no different.

Targeting TLR2 for Vaccine Development

Yes this is the title (Targeting TLR2 for Vaccine Dev) but using  Triacyl Lipoproteins for vaccine’s has already failed miserably causing the very disease they were supposed to protect against so best to avoid. See Dr Marks LymeRix FDA testimony Dr Marks FDA LymeRix Testimony .    There is no vaccine for TB or Syphilis because they are immune suppresssive diseases, and Lyme shouldn’t even be called Lyme, as the name was an attempt to seperate it out from other relapsing fever organisms  which is part of the crime, because it’s no different but they needed to market it with their narrow definition in order to carry on with their vaccine and test kit crimes (just another day at work for these psychopaths)

Braun’s lipoprotein from Escherichia coli is the prototype of the outer membrane triacylated lipoproteins from Gram-negative bacteria and some synthetic lipopeptides used as TLR2 stimulators, for example, tri-palmitoyl-S-glyceryl cysteine (Pam3C) SK4, have a lipid modi[cation analogue to this lipoprotein [48–50]. Other examples of triacylated lipoproteins are OspA from Borrelia burgdorferi [51] and the 19kDa lipoprotein from Mycobacterium tuberculosis [52, 53]. `e capacity to stimulate through TLR2 of both diacylated and triacylated lipoproteins is conferred by the lipidic N-terminal moiety [54, 55]. `e initial studies pointed that diacylated lipoproteins are signalized through TLR2/6 heterodimers, while the triacylated molecules do so through TLR2/1 heterodimers

These are some bad ass infections that all have immune suppression and PSS in common.

Mycobacterium tuberculosis 19-kilodalton lipoprotein inhibits Mycobacterium smegmatis-induced cytokine production by human macrophages in vitro

Moreover, lipoproteins evidently meet pathogen- associated molecular patterns (PAMPs) criteria and are well detected by innate immune recognition mechanisms [25]. M. tuberculosis lipoproteins are major antigens and trigger the activation of cellular and humoral immune responses to mycobacteria. Lipoproteins are potent agonists of toll-like receptor 2 (TLR2) which upon long term stimulation has been associated with the down regulation or deviation of the immune response.

Thus, the immunosuppressive effect is dependent on glycosylated and acylated 19-kDa lipoprotein present in the phagosome containing the mycobacterium. These results suggest that the diminished protection against challenge with M. tuberculosis seen in mice vaccinated with M. smegmatis expressing the 19-kDa lipoprotein is the result of reduced TNF-alpha and IL-12 production, possibly leading to reduced induction of T-cell activation.

So why did I add GWS and Vaccines to the list of PSS outcomes?  Well vaccines are often contaminated and GWS is likely caused by a cocktail of these vaccines. All it would take is a fungal contaminant of say a Mycobacteria or Mycoplasma to set off this chain reaction as well.    ME is another name for Post Sepsis Syndrome as is FMS and Autism.  They are just different presentations of the same mechanism.

Lipoproteins of Gram-Positive Bacteria: Key Players in the Immune Response and Virulence.

On the bacterial side, it has been shown that both S. aureus and the synthetic Lpp Pam2Cys and Pam3Cys alone induced severe bone loss in the femurs of mice after intraperitoneal (i.p.) administration and in a calvarial bone implantation model. However, the Δlgt mutant did not show such effects, indicating that Lpp are responsible for bone destruction during bacterial infections through augmentation of osteoclast differentiation and activation (35). Lpp also induce the inflammatory mediator nitric oxide (NO) in host cells. S. aureus and its mutants lacking lipoteichoic acid (ΔltaS) or D-alanylation of teichoic acids (ΔdltA) stimulated NO production in a murine macrophage cell line; however, the Δlgt mutant failed to induce NO production in a dose- dependent manner (36). These results suggest that not lipoteichoic acid (LTA) but Lpp of S. aureus induce NO production in murine macrophages through activation of TLR2. It is to be expected that Lpp in other pathogenic Gram-positive bacteria, such as Streptococcus pyogenes or Listeria monocytogenes, exert similar effects. S. aureus and synthetic Lpp, but not the Δlgt mutant, LTA, or peptidoglycan (PGN), induced IL-8 expression in the human intestinal epithelial cell-line Caco-2 (37).

Mycoplasma also causes PSS

A Triacylated Lipoprotein from Mycoplasma genitalium Activates NF-κB through Toll-Like Receptor 1 (TLR1) and TLR2

Of 10 TLR family members reported, TLR2, TLR4, TLR5, and TLR9 have been implicated in the recognition of different bacterial components. Peptidoglycan, lipoarabinomannan, zymosan, and lipoproteins from various microorganisms are recognized by TLR2 (2, 4, 22, 26, 46, 47, 49,52). On the other hand, lipopolysaccharide, bacterial flagellin, and bacterial DNA are recognized by TLR4, TLR5, and TLR9, respectively (12, 13, 16, 35). These TLR family members have been shown to activate NF-κB via interleukin-1R-associated signal molecules, including myeloid differentiation protein (MyD88), interleukin-1R-activated kinase, tumor necrosis factor receptor-associated factor 6, and NF-κB-inducing kinase (27)……………………………………………………………………………………………………………………………………………

“In this study, we examined the involvement of TLRs in activation of the immune response by lipoproteins from M. genitalium and their active components responsible for NF-κB activation. MG149, a triacylated lipoprotein, was found to activate NF-κB through TLR1 and TLR2.”

So to re-cap Post Sepsis is known to happen to 50% of Sepsis survivors (NIH)

PHYSICAL SYMPTOMS OF PSS:

• Lethargy/excessive tiredness
• Poor mobility / muscle weakness
• Breathlessness / chest pains
• Swollen limbs (excessive fluid in the tissues)
• Joint and muscle pains
• Insomnia
• Hair loss
• Dry / flaking skin and nails
• Taste changes
• Poor appetite
• Changes in vision
• Changes in sensation in limbs
• Repeated infections from the original site or a new infection
• Reduced kidney function
• Feeling cold
• Excessive sweating

PSYCHOLOGICAL AND EMOTIONAL SYMPTOMS OF PSS:

• Anxiety / fear of sepsis recurring
• Depression
• Flashbacks
• Nightmares
• Insomnia (due to stress or anxiety)
• PTSD (Post Traumatic Stress Disorder)
• Poor concentration
• Short term memory loss
• Mood swings

 

Since we know that structure predicts function, and we know the structure of Triacylated Lipoproteins, and we know where they bind (TLR 2/1), we can say with confidence that the same mechanism will cause the same outcome with all the diseases that shed them.   Immune Suppressive illnesses are the most brutally psychologically and physically abused groups in medicine, and these dominoes aren’t made of wood or plastic they are people, real people whose lives have meaning and purpose, and who deserve justice.

“Those who are capable of tyranny are capable of perjury to sustain it”

~Lysander Spooner

PROSECUTE POST SEPSIS/B CELL AIDS

FMS, CFS, ME, GWS, Autism, Vaccine induced = All in the same sinking boat

#OccupyJustice #JoinUs

For more info find us at TruthCures

 

 

The Psychological Warfare of the Medical Gaslighter

What is Gaslighting?

Inspired by the 1940 and 1944 films “Gas Light,” where a husband works to make his wife feel crazy by systematically manipulating her; “Gaslighting” is now commonly used to describe behavior that is utilized to manipulate someone’s reality.  You’re Not Going Crazy: 15 Signs You’re a Victim of Gaslighting

“The only way you can describe how you feel is that you feel minimized.  You feel crushed and smothered.  You’re constantly second-guessing yourself; your feelings, your perceptions, your memories, and a small, suffocated part inside of you wonders whether you are actually going crazy.”

If you don’t know you are under attack you are unlikely to be prepared to respond and this is what happens to patients every day who walk into their doctors offices in desperation; they are told in subtle or overt terms that its unlikely their symptoms are real or at any rate meaningful.  It’s very important to recognize when someone in a position of power is manipulating your reality.    Doctors will tell you straight up or allude to “it’s all in your head” for many reasons but none of them ever excuse this kind of behaviour and worse “all in your head” is the biggest lie they could tell you so it’s very important not to internalize this.

If I could give you one piece of advice its to decide ahead of time that you will never let anyone alter what you know to be true I don’t care how many medical degrees they have….

When It comes to medical gaslighting gender plays an important role and women are much more likely to be gaslit by their doctors.

How Doctors Take Women’s Pain Less Seriously

“Female pain might be perceived as constructed or exaggerated”: We saw this from the moment we entered the hospital, as the staff downplayed Rachel’s pain, even plain ignored it. In her essay, Jamison refers back to “The Girl Who Cried Pain,” a study identifying ways gender bias tends to play out in clinical pain management. Women are  “more likely to be treated less aggressively in their initial encounters with the health-care system until they ‘prove that they are as sick as male patients,’” the study concludes—a phenomenon referred to in the medical community as “Yentl Syndrome.”

Here’s one of the worst Lyme crooks Lenny Sigal, I will let you come to your own conclusions about him.

Gaslighting can be as simple as someone smirking at you and saying something like:

‘Gaslighting’ – or, why women are just too darned emotional during their heart attacks

• “You’re too sensitive!”
• “You’re so emotional!”
• “You’re defensive!”
• “You’re overreacting!”
• “Calm down!”
• “Just relax!”

I can’t tell you how many times I have been gaslit by doctors especially at the ER, in fact it’s so common that I believe they are trained in this method of keeping you unsure and off-balance so they can retain control.  A good example of subtle control methods is “medical rhetoric”  they have an entire language specifically created to ensure that only they have the full spectrum of information.  Why don’t they want patients to have full understanding about information pertaining to their body?  Either they don’t think we can handle the truth about our own health or they have another reason for keeping us in the dark.   The main purpose of language is shared understanding so when language takes on a cloak like this in my opinion it should always be questioned.  I am not talking about the terminology of the profession but the obvious communication style employed to cause a power differential between patient and doctor.

The eye roll, the blank stare, the scoffing sounds are very common gaslighting technique’s, once I was actually told by a Cardiologist and I quote “I don’t care about your symptoms”.   I repeated back to him “You don’t care about my symptoms?” and he again replied “no, not really.”   Now at this juncture we have two choices we can give our power away and slither out the office feeling completely inadequate or we can halt this disgusting behaviour in its tracks.    I recommend you chose B. As soon as the Cardiologist said this to me I stood up and said “Ok I think we’re done here” walked out of his office and made sure he saw me ask the receptionist for his card so he knew I was going to make an official complaint.   Within 5 mins he had called me to apologize and when I saw him in person he apologized again and said he had been reflecting on his behaviour and realized he was in the wrong.   Now this is a good outcome, there have been other times where it hasn’t worked out as well, but the way I see it if someone doesn’t believe what you are telling them you have nothing to lose anyway because if they aren’t for you, they are actually against you, so you might as well tell them that they are wrong and leave with your self-esteem intact.  And who knows it might make them reflect on their behaviour like it did for my Cardiologist.

We are Gaslit as individuals and we are also Gaslit as groups.   How long did it take for FMS, ME etc to be considered legitimate conditions? It’s also no accident that women are more likely to be diagnosed with these syndromes.  Lyme still isn’t considered a real or serious illness by most in the medical field.  Gaslighting can be employed to make the lives of those setting an agenda easier or it may have a much more nefarious agenda like in the case of Immune Suppressive illnesses.   Often times the doctor won’t really understand the full agenda but will have bought into the idea that some diseases and symptom clusters are just not real (or are psychological in nature), either way its best to stop BS in its tracks.

Robert Schoen another Lyme crook (researcher) doing all he can to put women who complain of lyme into the realm of psychiatric illness.

Lenny Sigal again singling out “women and girls” as if men and boys don’t get Lyme disease.

The powers that be (CDC etc) do not want us to recognize that we all suffer from immune suppressive illnesses (ME, Lyme, GWS, Autism, CFS) because that would take out millions of people from the vaccine market pool (cuz even the CDC says those with immune suppression shouldn’t get vaccines) plus it points an accusatory finger directly at them as their contaminated and fake vaccines are the CAUSE of many of these syndromes with Immune Suppressive outcomes.   They have basically set up every defensive strategy they can think of to block this information from making headway and often its psychological warfare that they employ. Think of what they have paid trolls to call Lyme patients “Lyme Loons” “Conspiracy Theorists”    We need people to recognize their power and the agendas of those who are doing all they can to take it away.

 

 

 

 

 

OspA, how we make em pay!

Click on the link below to view video titled “Chronic Lyme” What is the Illness to get an explanation of OspA by whistler blower Kathleen Dickson.

Kathleen Dickson explaining OspA

In the Lyme world OspA is the beginning of wisdom.

There is nothing the (CDC/ALDF) would like more than for us to continue believing that Lyme is just a bacterial infection that can be cured with antibiotics. In fact they would love it if we continued arguing over the false dichotomy, the fabricated battle between acute IDSA vs. chronic ILADS. The truth is neither of them have it right and we are wasting time fighting the wrong battles, spinning our wheels and getting nowhere. I don’t know about you but I get pretty mad when someone tries to pull the wool like this.

Lyme causes Post Sepsis Syndrome, it’s a B cell AIDS and just like HIV AIDS the initial infection isn’t what ends up doing the most damage its the immune suppression which leads to reactivations of latent viruses primarily the herpes viruses EBV, CMV etc and opportunistic infections that are the real culprits. The up side to this information is it matches our reality so well that you will likely see yourself very clearly in the following explanation and hopefully get relief from the chasing your tail game of treating infections ad infinitum and in vain. The other motivating factor is that there is no way any of us should allow the CDC/ALDF to get away with a medical genocide that has caused the suffering and deaths of millions of people – Selah (Pause and think on this).

By changing the case definition to exclude that vast majority of us with Neuro lyme and immune suppression and fraudulently removing OspA and B from testing they have deliberately left us on the side of the road to suffer and die. They committed this crime for the sole purpose of selling fake vaccine and test kits; kids dying so they can make a buck SMH. This is not some victimless crime this is a Medical Holocaust. I know OspA and permanent immune suppression isn’t a popular truth – I get that, it can be hard to come to terms with a piece of information like this, especially when many of us have believed if we treated long enough all would be well. However it IS the truth and denying it doesn’t change that so the best thing to do is learn the science as best you can, share the information and advocate for change that is based on the reality we live.

There is no celebrity waiting in the wings to save us, its just regular Joe’s like you and me and I for one do not want another generation of people completely guttered from this hideous disease.  So pony up!

OspA is an outter surface protein of the Spirochete that literally detonates and permanantly shuts down the immune system causing Post Sepsis Syndrome. The NIH agrees that 50% of people have immune suppression following a septic event.

TruthCures Justice in Healthcare

Carolyn Beans (NIH) on Live Science:

Surviving Sepsis: Detection and Treatment Advances

​Some people who survive sepsis can develop secondary infections days or even months later. A research team that included Richard Hotchkiss, Jonathan Green and Gregory Storch of Washington University School of Medicine in St. Louis suspected that this is because sepsis might cause lasting damage to the immune system. To test this hypothesis, the scientists compared viral activation in people with sepsis, other critically ill people and healthy individuals. The researchers looked for viruses like Epstein-Barr and herpes simplex that are often dormant in healthy people but can reactivate in those with suppressed immune systems. [Sepsis Has Long-Term Impact for Older Adults, Study Finds]

Of the three study groups, people with sepsis had much higher levels of these viruses, suggesting reactivation due to compromised immune responses. Immune suppression could make it difficult to defend against the reactivated viruses as well as new infections like pneumonia. The team now plans to test whether immune-boosting drugs can prevent deaths in sepsis survivors.”  Taken from Truthcures.org

PHYSICAL SYMPTOMS OF PSS:

• Lethargy/excessive tiredness
• Poor mobility / muscle weakness
• Breathlessness / chest pains
• Swollen limbs (excessive fluid in the tissues)
• Joint and muscle pains
• Insomnia
• Hair loss
• Dry / flaking skin and nails
• Taste changes
• Poor appetite
• Changes in vision
• Changes in sensation in limbs
• Repeated infections from the original site or a new infection
• Reduced kidney function
• Feeling cold
• Excessive sweating

PSYCHOLOGICAL AND EMOTIONAL SYMPTOMS OF PSS:

• Anxiety / fear of sepsis recurring
• Depression
• Flashbacks
• Nightmares
• Insomnia (due to stress or anxiety)
• PTSD (Post Traumatic Stress Disorder)
• Poor concentration
• Short term memory loss
• Mood swings

OspA is the mechanism and Post Sepsis Syndrome/ B cell AIDS is the outcome. Blebs or Osps that the Spirochete sheds are triacylated lipoproteins that are TLR 2/1 agonists. Anything that is a TLR 2/1 agonist sends the message to the immune system to shut down in order to prevent a deadly cytokine storm. Prior to this shut down the Septic event is in process and is causing a range of damage in the person depending on many factors such as virulence of the infection and the size and health of the person at the time of the event. A serious septic event can cause organ, vascular and cellular damage that can be permanent. These triacylated lipoproteins are so toxic that the body has no other option but to shut down in order to preserve life. While this is an adaptive response of the host it also causes many negative downstream effects such as immune suppression, tolerance and cross tolerance.

1-These results demonstrate that B. burgdorferi can stimulate the production of an antiinflammatory, immunosuppressive cytokine in naive cells and suggest that IL-10 may play a role both in avoidance by the spirochete of deleterious immune responses and in limiting the inflammation that the spirochete is able to induce.

Tolerance means that the immune system is no longer mounting a response to the pathogen, in this case borrelia burgdorferi (Bb) so in essence it has learned to ignore it, but that doesn’t mean it doesn’t continue to do damage because it certainly does. Cross tolerance is when the immune system also ignores other pathogens that bind to different TLR’s. For example OspA causes the immune system to ignore your common bacteria’s that normally bind to TLR 4 as well as viruses that bind to TLR 7 and 9 along with Borrelia itself (this is why if you get lyme a second or third time the immune system does nothing to combat it, it has not created memory cells from the previous exposure as the immune system normally would).

OspA is the main mechanism of the disease, we know this because you don’t actually need spirochetes to get all the symptoms of Neurological lyme all your need is OspA. Additionally, the first Lyme vaccine which was an OspA vaccine gave many people all the symptoms we associate with chronic Lyme disease.

Within 10 days of infection the germinal centres where B cells are assigned an immune duty have been shut down. B cells normally mature into antibody secreting cells, however in the presence of lyme they do not mature properly and therefore are functionally unable to produce antibodies needed to flag the immune system to respond causing B cell AIDS.  A permanently paralyzed immune system is a big deal that deserves real research attention and dollars and WE DESERVE JUSTICE!!! 

Post Sepsis Syndrome -Non HIV B Cell AIDS

~PROSECUTE POST SEPSIS~

#OccupyJustice # JoinUs

For more info check us out at TruthCures.org

REFERENCES:

1. Infect Immun. 1998 Jun;66(6):2691-7. Borrelia burgdorferi stimulates the production of interleukin-10 in peripheral blood mononuclear cells from uninfected humans and rhesus monkey

2. Sepsis Alliance Webpage.

3. TruthCures Justice In Healthcare

4.Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression.

5. Circulating bacterial membrane vesicles cause sepsis in rats